Atmospheric CO <sub xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink">2</sub> could potentially change ventilator rate leading to respiratory acidosis, and then daily life exposure can increase the intake rate of other air pollutants, therefore it's exposure dose. Subsequently CO <sub xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink">2</sub> may play an important role in regulating the toxicological effects of other air pollutants. In order to understand the possible effects of this pollutant on mammal's ventilatory rate, before designing experiments with living organisms, this study was carried out using a mathematical model that reproduces the main features of a chemosensitive neuron.